Renal anomalies: Renal Agenesis.. This illustration video deals with a common renal anomaly, Renal genesis.The facts have been presented in a way that makes clinical sense. In just 10 min whole topic has been dealt with ample detail.
Very use full, beautiful and highly comprehensive illustration with a lot of high yield facts and figures.
Bilateral renal agenesis is a condition in which both kidneys of a fetus fail to develop during gestation. It is one causative agent of Potter sequence. This absence of kidneys causes oligohydramnios, a deficiency of amniotic fluid in a pregnant woman, which can place extra pressure on the developing baby and cause further malformations. The condition is frequently, but not always the result of a genetic disorder, and is more common in infants born to one or more parents with a malformed or absent kidney. Males are more commonly affected and most infants that are born alive do not live beyond four hours
This is much more common, but is not usually of any major health consequence, as long as the other kidney is healthy.
It is associated with an increased incidence of Müllerian duct abnormalities, which are abnormalities of the development of the female reproductive tract and can be a cause of infertility, blocked menstrual flow (hematocolpos), increased need for Caesarean sections, or other problems. Herlyn-Werner-Wunderlich syndrome is one such syndrome in which unilaterial renal agenesis is combined with a blind hemivagina and uterus didelphys. Up to 40% of women with a urogenital tract anomaly also have an associated renal tract anomaly.
Adults with unilateral renal agenesis have considerably higher chances of hypertension (high blood pressure). People with this condition are advised to approach contact sports with caution.
Unilateral renal agenesis occurs approximately once in every 1000 newborn infants
Unilateral renal agenesis often causes no symptoms and is usually not discovered during infancy because the other
Bilateral renal agenesis is associated with oligohydramnios
infants have a characteristic facial appearance: the eyes are widely separated and have epicanthic folds, the ears are low-set, the nose is broad and flat, the chin is receding, and there are limb defects
metanephric diverticula fail to develop or the primordia of the ureters degenerate.
Failure of the metanephric diverticula to penetrate the metanephrogenic blastema results in failure of kidney development because no nephrons are induced by the collecting tubules to develop from the metanephrogenic blastemal
this sequence of anomalies is known as the Potter sequence
renal agenesis, hypoplasia, or dysfunction in utero is oligohydramnios
sloped forehead, “parrot beak” nose, shortened fingers, and hypoplasia of internal organs
large ears typical
Tags: Renal anomalies: Renal Agenesis., Kidney (Anatomical Structure), Renal Agenesis, compensatory hypertrophy, Potter sequence, parrot beak, oligohydramnios, hypoplasia, Oligohydramnios, metanephric diverticula, primordia of the ureters, polycystic, RENAL AGENESIS AND DYSPLASIA, amniotic fluid